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Patients requiring home parenteral nutrition (PN) may develop metabolic bone disease, the etiology of which can be multifactorial. We report a case of significantly low bone mass in a postmenopausal woman with history of short bowel syndrome, renal impairment, and previous radiation exposure who responded to intermittent subcutaneous administration of parathyroid hormone. Her bone mineral density normalized after she completed a course of 18 months of treatment, and a bone isotope scan was negative for skeletal malignancy.

Original publication

DOI

10.1177/0115426506021006605

Type

Journal article

Journal

Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition

Publication Date

12/2006

Volume

21

Pages

605 - 609

Addresses

University of Pennsylvania School of Medicine, 3615 Chestnut Street, Philadelphia, PA 19104, USA. pazianas@mail.med.upenn.edu

Keywords

Humans, Bone Diseases, Metabolic, Gastrointestinal Diseases, Teriparatide, Parenteral Nutrition, Home, Aged, Female, Bone Density Conservation Agents