Graphical abstract Establishment of an autoreactive B cell memory after myocardial infarction: a working hypothesis. The demise of cardiac cells leads to the release of cryptoantigens that induce a humoral immune response that leads to accumulation of immunoglobulins in plaques and eventually amplifies atherogenesis at remote sites.
Journal article
2021-03-01T00:00:00+00:00
42
948 - 950
2
Alarmins, Animals, Atherosclerosis, B-Lymphocytes, Immunoglobulins, Mice, Myocardial Infarction, Plasma Cells