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A theoretical model based upon a set of chemical reaction equations was established. It was assumed that activated LFA-1 cannot resume in passive or low affinity state as long as it is bound to ICAM-1. The initial results of the model suggest a role of active cellular processes (inside-out signaling) in LFA-1 activation.

Type

Journal article

Journal

Annals of biomedical engineering

Publication Date

01/12/2000

Volume

28