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Findings reported in The American Journal of Pathology suggest specialized proresolving mediators may become therapeutics that alleviate chronic tendon inflammation

Image shows interleukin IL-1beta stimulated tendon cells isolated from a patient with a shoulder tendon tear incubated in media containing either vehicle, 10nM Lipoxin B4 (LXB4), or 10nM Resolvin E1 (RvE1). Tendon cells were stained for inflammatory molecules pSTAT-1 (green) or IL-6 (red). LXB4 or RvE1 treatments moderate the pro-inflammatory phenotype of tendon cells, reducing expression of pSTAT-1 and IL6.

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Clinical and cost effectiveness of AI-enabled vertebral fracture identification Fracture Liaison Services (FLS)

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Vaccine LEGACY – Applying a systems based approach to accelerate vaccine development

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Harnessing human genomic, transcriptomic and proteomic data to identify novel therapeutic targets

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New pattern-recognition receptor signalling modalities in inflammation

Microbiome metabolites as immune checkpoints

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Resident memory B cell responses in the lung; immunity against pulmonary pathogens

How inflammatory macrophages instruct innate effector responses in the tumour microenvironment?

Engineering Resolution – mRNA targeting of chronic inflammatory disease

Investigating cross-tissue and cross-disease immune mechanisms using single-cell/spatial multiomics & machine learning/AI-powered language learning tools

Mechanical force matters in T-cell activation

Relationship between Tumour microenvironment and atherosclerotic cardiovascular disease during immunotherapy

Harnessing human genomic, transcriptomic and proteomic data to identify novel therapeutic targets

How do secreted phospho-proteins dictate fibroblast-neutrophil crosstalk in treatment resistant inflammatory bowel disease?

Unraveling the intersection of immune responses in gut and cardiovascular and health

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Proresolving Mediators LXB4 and RvE1 Regulate Inflammation in Stromal Cells from Patients with Shoulder Tendon Tears

 

 

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