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Search results (13)

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In silico degradomics reveals disease- and endotype-specific alterations in the joint tissue landscape

Hoyle A. and Midwood KS., (2026)

Biomechanical and compositional basement membrane defects due to a Col4a1 mutation affect cardiac morphology and function.

Boland E. et al, (2025), Matrix Biol, 141, 82 - 100

Synovial matrix turnover controls immune cell spatial patterning in inflammation resolution.

Richard J-B. et al, (2025), Mol Syst Biol, 21, 1638 - 1665

Multiple golgins are required to support extracellular matrix secretion, modification, and assembly.

Thompson G. et al, (2025), J Cell Biol, 224

Endocytic recycling is central to circadian collagen fibrillogenesis and disrupted in fibrosis.

Chang J. et al, (2025), Elife, 13

Collagen fibril formation at the plasma membrane occurs independently from collagen secretion.

Pickard A. et al, (2025), Wellcome Open Res, 10

Degradomics defines proteolysis information flow from human knee osteoarthritis cartilage to matched synovial fluid and the contributions of secreted proteases ADAMTS5, MMP13 and CMA1 to articular cartilage breakdown.

Bhutada S. et al, (2025), Osteoarthritis Cartilage, 33, 116 - 127

Collagen fibril formation at the plasma membrane occurs independently from collagen secretion.

Pickard A. et al, (2024), bioRxiv

Golgins support extracellular matrix secretion by collectively maintaining the Golgi structure-function relationship

Thompson G. et al, (2024)

Circadian and permanent pools of extracellular matrix co-exist in tendon tissue, but have distinct rates of turnover and differential responses to ageing

Hoyle A. et al, (2024)

Optimising a self-assembling peptide hydrogel as a Matrigel alternative for 3-dimensional mammary epithelial cell culture

Lingard E. et al, (2023)

SGOL1-AS1 enhances cell survival in acute myeloid leukemia by maintaining pro-inflammatory signaling.

Selkirk E. et al, (2022), Heliyon, 8

ER-Golgi and lysosomal pathways together drive fibrous tissue formation

Pickard A. et al, (2022)