Publications

Autoimmunity-associated T cell receptors recognize HLA-B*27-bound peptides.

Yang X. et al, (2022), Nature

Constructing custom-made radiotranscriptomic signatures of vascular inflammation from routine CT angiograms: a prospective outcomes validation study in COVID-19.

Kotanidis CP. et al, (2022), Lancet digit health

A blood atlas of COVID-19 defines hallmarks of disease severity and specificity.

COvid-19 Multi-omics Blood ATlas (COMBAT) Consortium. Electronic address: julian.knight@well.ox.ac.uk None. and COvid-19 Multi-omics Blood ATlas (COMBAT) Consortium None., (2022), Cell, 185, 916 - 938.e58

An immunodominant NP105-113-B*07:02 cytotoxic T cell response controls viral replication and is associated with less severe COVID-19 disease.

Peng Y. et al, (2022), Nat immunol, 23, 50 - 61

FUNCTIONAL GENOMICS INVESTIGATION OF THE ANKYLOSING SPONDYLITIS ASSOCIATED LOCUS RUNX3

Cohen C. et al, (2022), Annals of the rheumatic diseases, 81, 231 - 231

Intrinsic Folding Properties of the HLA-B27 Heavy Chain Revealed by Single Chain Trimer Versions of Peptide-Loaded Class I Major Histocompatibility Complex Molecules.

Lenart I. et al, (2022), Front immunol, 13

Single cell analysis of spondyloarthritis regulatory T cells identifies distinct synovial gene expression patterns and clonal fates.

Simone D. et al, (2021), Commun biol, 4

Ex vivo mass cytometry analysis reveals a profound myeloid proinflammatory signature in psoriatic arthritis synovial fluid.

Yager N. et al, (2021), Ann rheum dis, 80, 1559 - 1567

SINGLE CELL ANALYSIS OF SPONDYLOARTHRITIS REGULATORY T CELLS IDENTIFIES DISTINCT SYNOVIAL GENE EXPRESSION PATTERNS AND CLONAL FATES

Simone D. et al, (2021), Annals of the rheumatic diseases, 80, 17 - 18

A blood atlas of COVID-19 defines hallmarks of disease severity and specificity

Ahern DJ. et al, (2021)

Ex vivo mass cytometry analysis reveals a profound myeloid proinflammatory signature in psoriatic arthritis synovial fluid

Yager N. et al, (2021)

A FIRST IN DISEASE PHASE 2A TRIAL OF GRANULOCYTE MONOCYTE COLONY STIMULATING FACTOR NEUTRALISATION FOR AXIAL SPONDYLOARTHRITIS (NAMASTE STUDY)

Al-Mossawi MH. et al, (2021), Clinical and experimental rheumatology, 39, 1161 - 1161

COMPREHENSIVE EPIGENOMIC PROFILING REVEALS DISEASE-SPECIFIC CHROMATIN STATES IN ANKYLOSING SPONDYLITIS

Cohen CJ. et al, (2021), Clinical and experimental rheumatology, 39, 1155 - 1155

Disruption of c-MYC Binding and Chromosomal Looping Involving Genetic Variants Associated With Ankylosing Spondylitis Upstream of the RUNX3 Promoter.

Cohen CJ. et al, (2021), Front genet, 12

From the Genetics of Ankylosing Spondylitis to New Biology and Drug Target Discovery.

Nancy Z. et al, (2021), Front immunol, 12

NOVEL USE OF SPECIFIC DRUGS ATTENUATES THE PRO-INFLAMMATORY CYTOKINES IL-17A AND IFN-GAMMA

Ridley A. et al, (2021), Clinical and experimental rheumatology, 39, 1164 - 1164

PROINFLAMMATORY OSTEOPONTIN AND CCL2 ARE SPONTANEOUSLY PRODUCED BY PSORIATIC ARTHRITIS SYNOVIAL MYELOID CELLS

Yager N. et al, (2021), Clinical and experimental rheumatology, 39, 1212 - 1212

Single-cell sequencing reveals clonal expansions of pro-inflammatory synovial CD8 T cells expressing tissue-homing receptors in psoriatic arthritis.

Penkava F. et al, (2020), Nat commun, 11

New Developments in our Understanding of Ankylosing Spondylitis Pathogenesis

BOWNESS P. and Voruganti A., (2020), Immunology

Identification of an Unconventional Subpeptidome Bound to the Behçet's Disease-associated HLA-B*51:01 that is Regulated by Endoplasmic Reticulum Aminopeptidase 1 (ERAP1).

Chen L. et al, (2020), Mol cell proteomics, 19, 871 - 883

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Publications