A longitudinal single-cell atlas of anti-tumour necrosis factor treatment in inflammatory bowel disease.
Thomas T. et al, (2024), Nat immunol, 25, 2152 - 2165
MHC class II antigen presentation by intestinal epithelial cells fine-tunes bacteria-reactive CD4 T-cell responses.
Heuberger CE. et al, (2024), Mucosal immunol, 17, 416 - 430
Elevated type-17 cytokines are present in Axial Spondyloarthritis stool
Brough I. et al, (2024), Discovery immunology
Blimp-1 and c-Maf regulate immune gene networks to protect against distinct pathways of pathobiont-induced colitis.
Alvarez-Martinez M. et al, (2024), Nat immunol
Alterations in the gut microbiome implicate key taxa and metabolic pathways across inflammatory arthritis phenotypes.
Thompson KN. et al, (2023), Sci transl med, 15
Adipocyte autophagy limits gut inflammation by controlling oxylipin and IL-10.
Richter FC. et al, (2023), Embo j
Fates of Commensal Specific, Innate-like CD8+T cells in the Gut
Farley TK. et al, (2023), Journal of immunology, 210
A conserved population of MHC II-restricted, innate-like, commensal-reactive T cells in the gut of humans and mice.
Hackstein C-P. et al, (2022), Nat commun, 13
Hermansky-Pudlak syndrome type 1 causes impaired anti-microbial immunity and inflammation due to dysregulated immunometabolism.
Cavounidis A. et al, (2022), Mucosal immunol
A conserved population of MHC II-restricted, innate-like, commensal-reactive T cells in the gut of humans and mice
Hackstein C-P. et al, (2022)
Cross-tissue, single-cell stromal atlas identifies shared pathological fibroblast phenotypes in four chronic inflammatory diseases.
Korsunsky I. et al, (2022), Med (n y), 3, 481 - 518.e14
Author Correction: Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease.
Serra EG. et al, (2022), Nat commun, 13
Genetic and environmental factors shape the host response to Helicobacter hepaticus: insights into IBD pathogenesis.
Jeffery R. et al, (2022), Curr opin microbiol, 65, 145 - 155
IL-1-driven stromal-neutrophil interactions define a subset of patients with inflammatory bowel disease that does not respond to therapies.
Friedrich M. et al, (2021), Nat med, 27, 1970 - 1981
Adipocyte autophagy limits gut inflammation by controlling oxylipin levels
Richter FC. et al, (2021)