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Interleukin-15-activated natural killer cells kill autologous osteoclasts via LFA-1, DNAM-1 and TRAIL, and inhibit osteoclast-mediated bone erosion in vitro.
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Interleukin-15-activated natural killer cells kill autologous osteoclasts via LFA-1, DNAM-1 and TRAIL, and inhibit osteoclast-mediated bone erosion in vitro
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Balance between activating NKG2D, DNAM-1, NKp44 and NKp46 and inhibitory CD94/NKG2A receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.
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AB0079 Synovial Explant Inflammatory Mediator Production is Correlated with Disease Activity in Rheumatoid Arthritis: A Cross Sectional Study
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FRI0021 Synovial Explant Activity is Associated with Disease Activity in Biologically Treated Rheumatoid Arthritis Patients: A Cohort Study
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Synovial explant inflammatory mediator production corresponds to rheumatoid arthritis imaging hallmarks: a cross-sectional study.
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Synovial Explant Inflammatory Mediator Production Is Associated With Synovitis While Not With Bone Marrow Edema In Rheumatoid Arthritis: A Cross Sectional Study
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FRI0020 Blocking the inhibitory CD94/NKG2A NK cell receptor with a novel ANTI-NKG2A MAB enhances the susceptibility of rheumatoid arthritis fibroblast-like synoviocytes (FLS) to NK cell-mediated cytotoxicity
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OP0142 Masking CD94/NKG2A using a novel therapeutic MAB results in significant suppression of IL-6 levels and reduced osteoclast formation in rheumatoid arthritis ex vivo cultures
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T(H)1, T(H)2, and T(H)17 cells instruct monocytes to differentiate into specialized dendritic cell subsets.
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Activating and inhibitory receptors on synovial fluid natural killer cells of arthritis patients: role of CD94/NKG2A in control of cytokine secretion.
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Natural killer cells trigger differentiation of monocytes into dendritic cells.
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Qa-1(b)-dependent modulation of dendritic cell and NK cell cross-talk in vivo.
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Qa-1(b)-Dependent modulation cross-talk in vivo
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