Identification of an immunodominant peptide from citrullinated tenascin-C as a major target for autoantibodies in rheumatoid arthritis.
Schwenzer A. et al, (2016), Ann rheum dis, 75, 1876 - 1883
Crystal structure of Porphyromonas gingivalis peptidylarginine deiminase: implications for autoimmunity in rheumatoid arthritis.
Montgomery AB. et al, (2016), Ann rheum dis, 75, 1255 - 1261
Citrullination in the periodontium--a possible link between periodontitis and rheumatoid arthritis.
Laugisch O. et al, (2016), Clin oral investig, 20, 675 - 683
Antibodies to Porphyromonas gingivalis Indicate Interaction Between Oral Infection, Smoking, and Risk Genes in Rheumatoid Arthritis Etiology.
Kharlamova N. et al, (2016), Arthritis rheumatol, 68, 604 - 613
Erratum to: Smoking, Porphyromonas gingivalis and the immune response to citrullinated autoantigens before the clinical onset of rheumatoid arthritis in a Southern European nested case-control study.
Fisher BA. et al, (2016), Bmc musculoskelet disord, 17
Citrullination-acetylation interplay guides E2F-1 activity during the inflammatory response.
Ghari F. et al, (2016), Sci adv, 2
Smoking, Porphyromonas gingivalis and the immune response to citrullinated autoantigens before the clinical onset of rheumatoid arthritis in a Southern European nested case-control study.
Fisher BA. et al, (2015), Bmc musculoskelet disord, 16
Immunomodulation By a Second-Generation Peptidyl Arginine Deiminase Inhibitor Abrogates Collagen-Induced Arthritis in a Therapeutic Protocol
Quirke A-M. et al, (2015), Arthritis & rheumatology, 67
Bronchiectasis is a Model for Chronic Bacterial Infection Inducing Autoimmunity in Rheumatoid Arthritis.
Quirke A-M. et al, (2015), Arthritis rheumatol, 67, 2335 - 2342
Expression of citrulline and homocitrulline residues in the lungs of non-smokers and smokers: implications for autoimmunity in rheumatoid arthritis.
Lugli EB. et al, (2015), Arthritis res ther, 17
PPAD remains a credible candidate for inducing autoimmunity in rheumatoid arthritis: comment on the article by Konig et al.
Quirke A-M. et al, (2015), Ann rheum dis, 74
Response to: 'The autoantibody repertoire in periodontitis: a role in the induction of autoimmunity to citrullinated proteins in rheumatoid arthritis? Antibodies against uncitrullinated peptides seem to occur prior to the antibodies to the corresponding citrullinated peptides' by Brink et al.
de Pablo P. et al, (2014), Ann rheum dis, 73
SMOKING AFFECTS IMMUNE RESPONSE TO CITRULLINATED AUTOANTIGENS BEFORE CLINICAL ONSET OF RHEUMATOID ARTHRITIS IN A SOUTHERN EUROPEAN COHORT
Cartwright AJ. et al, (2014), Rheumatology, 53, 36 - 36
A1.5 Smoking is a risk factor for ACPA prior to onset of symptoms of rheumatoid arthritis in a cohort from southern europe.
Cartwright AJ. et al, (2014), Ann rheum dis, 73 Suppl 1, A2 - A3
The autoantibody repertoire in periodontitis: a role in the induction of autoimmunity to citrullinated proteins in rheumatoid arthritis?
de Pablo P. et al, (2014), Ann rheum dis, 73, 580 - 586
Heightened immune response to autocitrullinated Porphyromonas gingivalis peptidylarginine deiminase: A potential mechanism for breaching immunologic tolerance in rheumatoid arthritis
Quirke AM. et al, (2014), Annals of the rheumatic diseases, 73, 263 - 269
Heightened immune response to autocitrullinated Porphyromonas gingivalis peptidylarginine deiminase: a potential mechanism for breaching immunologic tolerance in rheumatoid arthritis.
Quirke A-M. et al, (2014), Ann rheum dis, 73, 263 - 269
The case for measuring antibodies to specific citrullinated antigens.
Montgomery AB. et al, (2013), Expert rev clin immunol, 9, 1185 - 1192
Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD).
Maresz KJ. et al, (2013), Plos pathog, 9
SAT0110 Is periodontal disease a risk factor for rheumatoid arthritis? The anti-citrullinated antibody repertoire in periodontal disease
de Pablo P. et al, (2013), Annals of the rheumatic diseases, 71, 507.3 - 507