Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Lymphocyte function-associated-antigen-1 (LFA-1) is able to bind selectively to its ligands intercellular adhesion molecules 1 and 3 (ICAM-1 and ICAM-3), suggesting that LFA-1 can exist in distinct ligand-specific binding states. In the case of ICAM-1, apart from ligand itself and the recently cloned molecule cytohesin-1, the natural physiological regulators of LFA-1-mediated binding to ICAM-1 are unknown. We have investigated the role of ligands (ICAM-1 and ICAM-3) in LFA-1 activation by using ICAM-blocking monoclonal antibodies and a fixation protocol for "freezing" LFA-1 on the surface of cells after prior exposure to ICAM-1 and ICAM-3. These studies not only confirm that LFA-1 exists in distinct ICAM-specific activation states, but also demonstrate that ICAM-1 plays a role in the activation of LFA-1 binding to ICAM-3.

Original publication

DOI

10.1002/eji.1830270423

Type

Journal article

Journal

Eur j immunol

Publication Date

04/1997

Volume

27

Pages

957 - 962

Keywords

Antigens, CD, Antigens, Differentiation, Cell Adhesion, Cell Adhesion Molecules, Fixatives, Formaldehyde, Humans, Intercellular Adhesion Molecule-1, Leukemia, Erythroblastic, Acute, Ligands, Lymphocyte Function-Associated Antigen-1, Polymers, Protein Binding, Tumor Cells, Cultured